Alzheimer’s Plaques Co-Accumulate with Potential Biomarker or Target Proteins

Amyloid beta deposits, long linked to Alzheimer’s disease (AD), have been found by Emory University scientists to aid the accumulation of other proteins in the brain. Rather than the amyloid deposits, it is these proteins, with known signalling functions, that may cause the brain cell damage associated with AD. The proteins co-accumulate with amyloid beta in both human brains with Alzheimer’s and mice, suggesting potential as new therapeutic targets.